The Collins Lab
Xiong X., Hao, Y., Sun, K., Li., J., Li, X., Mishra, B., Soppina, P., Wu, C., Hume, R. I. and Collins, C.A.. (2012). "The Highwire ubiquitin ligase promotes Wallerian degeneration by tuning levels of Nmnat protein." PLos Biology. In press.

Ghanned-Razaie, M.*, Wang, X.*, Mishra, B*., Collins, C.A.**, and Chronis, N.**. (2012). "Microfluidic chips for in vivo imaging of cellular responses to neural injury in Drosophila larvae." PLos One, 7(1):e29869. *co-first authors, **co-corresponding authors. PubMed

Xiong, X., and Collins, C.A.. (2012). "A Conditioning Lesion Protects Axons from Degeneration via the Wallenda/DLK MAP Kinase Signaling Cascade." Journal of Neuroscience, Jan 11;32(2):610-5. PubMed

Xiong X, Wang X, Ewanek R, Bhat P, Diantonio A, and Collins C.A.. (2010). "Protein turnover of the Wallenda/DLK kinase regulates a retrograde response to axonal injury." Journal of Cell Biology, 191:211-23. PubMed

Horiuchi, D.*, Collins, C.A.*, Barkus, R.V., Bhat, P., DiAntonio, A., and Saxton, W.M.. (2007). "Control of a kinesin-cargo linkage mechanism by JNK pathway kinases." Current Biology, 17:1313-7. *co-first author. PubMed

Collins, C.A., and DiAntonio, A.. (2007). "Synaptic development: insights from Drosophila." Current Opinion in Neurobiology, 17: 35-42. PubMed

Collins, C.A., Wairkar, Y.P., Johnson, S.L. and DiAntonio, A.. (2006). "Highwire restrains synaptic growth by attenuating a MAP kinase signal." Neuron, 51: 57-69. PubMed

Daniels R.W., Collins C.A., Chen, K., Gelfand, M.V., Featherstone, D.E., DiAntonio, A.. (2006). "A single vesicular glutamate transporter is sufficient to fill a synaptic vesicle." Neuron, 49: 11-6. PubMed

Wu, C., Wairkar Y.P., Collins C.A., DiAntonio A.. (2005). "Highwire function at the Drosophila neuromuscular junction: spatial, structural, and temporal requirements." Journal of Neuroscience, 25: 9557-66. PubMed

Daniels, R.W.*, Collins, C.A*., Gelfand, M.V., Dant, J., Brooks, E.S., Krantz, D.E., DiAntonio, A.. (2004). "Increased expression of the Drosophila vesicular glutamate transporter leads to excess glutamate release and a compensatory decrease in quantal content." Journal of Neuroscience, 24: 10466-74. *Co-first author. PubMed

Collins, C. A., and DiAntonio, A.. (2004). "Coordinating synaptic growth without being a nervous wreck." Neuron, 41: 489-91. PubMed

Collins, C. A., and Guthrie, C.. (2001). "Genetic interactions between the 5' and 3' splice site consensus sequences and U6 snRNA during the second catalytic step of pre-mRNA splicing." RNA, 7: 1845-54. PubMed

Collins, C. A., and Guthrie, C.. (2000). "The question remains: Is the spliceosome a ribozyme?." Nature Structual Biology, 7: 850-4. PubMed

Collins, C. A., and Guthrie, C.. (1999). "Allele-specific genetic interactions between Prp8 and RNA active site residues suggest a function for Prp8 at the catalytic core of the spliceosome." Genes and Development, 13: 1970-1982. PubMed